Not known Factual Statements About pro33
Not known Factual Statements About pro33
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To better understand the molecular mechanisms by which this sequence variation modifies platelet function, we created transgenic knockin mice expressing a Pro32Pro33 integrin β
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A number of with the phenotypes presented in Fig. 2 were examined in the context of protease-activated receptor activation by thrombin or PAR4-AP. While our facts disclosed altered outside the house-in signaling in unstimulated platelets, a number of reports have delivered evidence for agonist-induced improvement of FAK and ERK signaling in platelets and cells expressing the human Pro33 integrin β
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